Menopause Hormone Blood Test

Menopause Hormone Blood Test

August 26, 2011

The Menopause Hormone Blood Test identifies deficiencies (or excesses) of sex hormones estradiol, estrone, estriol, progesterone, and testosterone.

The “female” estrogens: estriol, estradiol, and estrone, all affect health throughout the body.

Progesterone, which balances the action of estrogen, serves as a precursor for other hormones, and plays an important role in mood, blood sugar balance, libido, and thyroid function, as well as adrenal gland health.

The “male” hormone testosterone, which helps to maintain lean body mass, bone density, skin elasticity, blood cell production, and libido .

Health conditions this test is used to assess
This test can determines your baseline hormone levels, so you can make an informed decision about hormone replacement therapy. It can also provide guidance in the treatment of endometriosis , breast cancer, high blood pressure and heart disease, osteoporosis, and low libido.

What this test involves
This test involves collecting three saliva samples collected in test tubes and sent to the lab.

How can I get this test done?
Talk to your health care professional about your symptoms and ask if this test would be useful for you

Required Male Blood Panel for Hormone Replacement Therapy

1. Homocystine,Plasma……
2. TSH Free T4
3. Lipid Panel With LDL/HDL Ratio
4. Comp. Metabolic Panel
5. Testosterone Free and Total
6. IGF-1
7. Triiodothyronine, Free, Serum
8. FSH, LH
9. Estradiol
10. CBC With Differential/Platelet
11. PSA
12. Cortisol
13. DHEA Sulfate
14. Hemoglobin A1C
15. Thyroid Panel with TSH
16. Insulin, Fasting
17. Ferritin, Serum
18. SHBG

10 reasons for Hormone Replacement Therapy (HRT) Clinic

10 reasons for Hormone Replacement Therapy (HRT) Clinic

August 15, 2011
(1). HRT will stop your hot flushes and sweats

Troublesome hot flushes, severe night sweats and headaches causingchronic insomnia are characteristic symptoms of the menopause.These symptoms may last for many years. Apart from being sociallyembarrassing they result in tiredness and depression becauseof lack of sleep. These symptoms can almost invariably be curedwith the correct small dose of estrogen. Although selectiveserotonin reuptake inhibitor antidepressants have been suggestedfor the treatment of vasomotor symptoms, no other treatmentis nearly as effective as estrogens. Women who still have auterus should still have 7–12 days of progestogen in orderto produce a withdrawal bleed and prevent endometrial hyperplasia.

(2). Estrogens will treat vaginal dryness and many causes of painful intercourse and lack of libido

Thinning of the pelvic tissues producing vaginal dryness andoccasionally bleeding is another characteristic result of estrogendeficiency that occurs after the menopause. This also can besuccessfully treated with estrogen either by tablets or throughthe skin by patches or gels or implants. Transdermal estrogentherapy is probably the safest and most effective route as hepaticcoagulation factors are not stimulated. Local estrogens canalso be given for this symptom using local vaginal applicationsof weak estrogens such as oestriol that are hardly absorbed.Other related problems of painful intercourse, loss of libidoand recurrent ‘cystitis’, if due to pelvic atrophyare also effectively treated by systemic or long-term localvaginal estrogens.

(3). HRT increases bone density and prevents osteoporotic fractures

Every study confirms that estrogens are the most effective wayof increasing bone density and preventing osteoporotic fractureseven in low-risk women. This treatment is very safe when startedin women under the age of 60. It is more effective and beneficialthan the bisphosphonates that are frequently used by bone physiciansas first choice and by general practitioners unsure about thesafety of estrogen therapy. These non-hormonal drugs with theirconsiderable long-term complications should have no place inmaintaining bone density in women under the age of 60. For therecently menopausal women receiving estrogen therapy for climactericsymptoms such as flushes, sweats or vaginal dryness, there willbe a considerable increase, up to 15% in 10 years to such anextent that osteoporotic fractures 20 years later in the olderwomen are much less likely to occur. If these women have lowbone density, even without typical menopausal symptoms, estrogensmust be seen as first-choice therapy. For those younger womenwith severe osteopenia or osteoporosis due to premature menopause,early hysterectomy and oophorectomy or anorexia with amenorrhoea,estrogens are an essential long-term treatment.

(4). HRT protects the intervertebral discs

Important recent studies from several centres have shown conclusivelythat estrogens prevent collagen being lost from the intervertebraldiscs, thus maintaining their strength and function. These discsmake up one-quarter of the length of the spinal column and actas cushions preventing crush fractures of the vertebral bodies.It is these crush fractures that lead to loss of height andthe lordosis of the upper spine known as the Dowager’s hump.This important protective effect of estrogens seems to be uniqueas bisphosphonates and the other non-hormonal treatments oflow bone density do not have any beneficial effect upon thediscs. {As pointed out elsewhere bisphosphonates increase bone density by going to the bond long-term, but done make the bones more resistant to fractures–jk}.

(5). HRT does reduce the number of heart attacks

There are about 30 years of evidence from many observationaltrials that estrogens reduce the incidence of coronary heartdisease. This has subsequently been questioned by the 2002 WHIStudy, which showed an increase in heart attacks. However, thisstudy looked at patients of the wrong age and who were usingthe wrong dose of estrogen and progestogen. Subsequent reportsfrom the same investigators have shown a very much reduced incidenceof heart attacks in women who start HRT below the age of 60.This is particularly apparent in women who have had a hysterectomyand can have estrogens without progestogen. The view now isthat HRT, particularly estrogen alone, is very safe and is associatedwith a reduced number of heart attacks if started below theage of 60. Thus there is primary prevention of coronary heartdisease, but there is no evidence of protection in women withestablished coronary damage.

It would appear that the factor that is associated with theapparent increase in severe side-effects such as breast cancerand heart attacks and possibly stroke is the progestogen componentof HRT. As progestogen also produces unwanted PMS-type side-effectsof depression, anxiety, bloating and loss of libido in patientswho are progestogen intolerant, it is sensible to keep the doseof oral gestogen to a minimum. The alternative is to inserta Mirena intrauterine system, which produces amenorrhoea andavoids the use of oral progestogen with its side-effects forfive years or more.

(6). Estrogens help depression in many women

Estrogens are more effective in the treatment of depressionin premenopausal or perimenopausal women than post- menopausalwomen. However there is no doubt that depression is helped inpostmenopausal women who have been suffering from night sweats,insomnia or vaginal dryness, painful intercourse and maritalproblems in that most of these problems can be effectively treatedand removed. However, it is true that the most impressive effecton mood is seen in younger perimenopausal women in the 2–3years before the period cease in the menopausal transition.This cannot be diagnosed by blood tests but by a careful history.This depression often occurs in women who are sensitive to abruptchanges in their hormones, either endogenous oestradiol or progesterone.These women had previously had postnatal depression and premenstrualdepression in what should be known as reproductive depression.They often also have cyclical headaches/migraines that occurwith the cyclical hormonal fluctuations at menstruation. Aspremenstrual depression becomes worse with age, it blends intothe more severe depression of the transition phase and is veryeffectively treated by moderately high-dose transdermal estrogensused by patches, gels or implants.

(7). HRT improves libido

HRT certainly improves libido if estrogens are used to curevaginal dryness and painful intercourse. Even without thesecharacteristic symptoms, estrogens can improve sexual desire.However, if necessary, the addition of testosterone has a moredramatic effect upon libido, frequency of intercourse and intensityof orgasm. Testosterone patches licensed in women after hysterectomyand testosterone gels in the appropriate dose are often andshould be used ‘off license’ with full consent andexplanation.

Women must be aware that testosterone is not only a male hormonebut it is an essential female hormone present in women in about10 times the blood levels as estrogen. It is an essential hormone,important for energy, mood and sexuality.

(8). HRT improves the texture of the skin

After the menopause, women lose about 25% of their body collagen,which is manifested by thin inelastic skin, brittle nails, lossof hair and loss of the collagenous bone matrix. This latterloss is an essential cause of osteoporosis and osteoporoticfractures. Estrogen therapy replaces the lost collagen in theskin and the bone. Its affect on the facial skin is a very obvioususeful cosmetic effect.

(9). ‘I am a nicer person to live with’

This is a quote from a patient. Many women say that when estrogentherapy stops their depression, their loss of libido and theirirritability, they become more agreeable people for their partnersto live with. The depression, irritability, grumpiness and lossof energy and disinterest in sex can usually be improved considerablyby the appropriate doses of the appropriate hormones that mayinclude testosterone as well as estrogen.

(10). HRT is safe

In spite of the press reports stressing bad news, virtuallyall claims of major adverse effects from the WHI study havebeen reconsidered even by the investigators. It seems quiteclear that the reported major side-effects of breast cancer,stroke and heart attacks occurred in women who started the wrongdose of HRT over the age of 60. In women who started below theage of 60 there were fewer heart attacks, fewer deaths, fewerosteoporotic fractures and even less breast cancer in this study.It is probable that the one residual side-effect is a small1% extra lifetime risk of developing breast cancer, but thisis no more than the breast cancer risk of being overweight,drinking wine, having no children or even taking statins. {DISAGREE: As above stated the author

Low Testosterone – what causes it?

Low Testosterone – what causes it?

August 5, 2011

What causes testosterone deficiency?

Testosterone is a hormone produced by the testicles and is responsible for the proper development of male sexual characteristics, and is important for maintaining muscle bulk, adequate levels of red blood cells, bone density, sense of well-being, and sexual and reproductive function.

Inadequate testosterone production is not a common cause of erectile dysfunction (ED). When ED does occur with decreased testosterone production, testosterone replacement therapy may improve the ED.

As a man ages, the amount of testosterone in his body gradually declines. This natural decline starts after age 30 and continues throughout life. The significance of this decline is controversial and poorly understood.

Symptoms of testosterone deficiency:

  • decreased sex drive
  • decreased sense of well-being
  • depressed mood
  • difficulties with concentration and memory
  • erectile dysfunction

What are the changes that occur in the body with testosterone deficiency?

Changes that occur with testosterone deficiency include:

  • a decrease in muscle mass, with an increase in body fat
  • variable effects on cholesterol metabolism
  • a decrease in hemoglobin and possibly mild anemia
  • fragile bones (osteoporosis)
  • a decrease in body hair

How do I find out if I have a testosterone deficiency?

The only accurate way to detect the condition is to have your doctor measure the amount of testosterone in your blood. It sometimes may take several measurements of testosterone to be sure if a patient has a deficiency, since levels of testosterone tend to fluctuate throughout the day. The highest levels of testosterone are generally in the morning. This is why doctors prefer, if possible, to obtain early morning levels of testosterone.

What options are available for testosterone replacement?

The options available for testosterone replacement are:

  • intramuscular injections, generally every two or three weeks
  • testosterone patches worn either on the body or on the scrotum (the sac that contains the testicles). These patches are used daily. The body patch application is rotated between the buttocks, arms, back or abdomen.
  • testosterone gels that are applied daily to the shoulders, upper arms, or abdomen.

For a free consultation on what would work best for you, contact us at: or call us at 866-641-CORE (2673)



August 4, 2011

Endogenous androgens are responsible for normal growth and development of the male sex organs and for maintenance of secondary sex characteristics. These effects include growth and maturation of the prostate, seminal vesicles, penis, and scrotum; development of male hair distribution, such as beard, pubic, chest, and axillary hair, laryngeal enlargement, vocal cord thickening, and alterations in body musculature and fat distribution. Drugs in this class also cause retention of nitrogen, sodium, potassium, and phosphorous, and decreased urinary excretion of calcium. Androgens have been reported to increase protein anabolism and decrease protein catabolism. Nitrogen balance is improved only when there is sufficient intake of calories and protein.

Androgens are responsible for the growth spurt of adolescence and for eventual termination of linear growth, brought about by fusion of the epiphyseal growth centers. In children, exogenous androgens accelerate linear growth rates, but may cause disproportionate advancement in bone maturation. Use over long periods may result in fusion of the epiphyseal growth centers and termination of the growth process. Androgens have been reported to stimulate production of red blood cells by enhancing production of erythropoietic stimulation factor.

During exogenous administration of androgens, endogenous testosterone release is inhibited through feedback inhibition of pituitary luteinizing hormone (LH). At large doses of exogenous androgens, spermatogenesis may also be suppressed through feedback inhibition of pituitary follicle stimulating hormone (FSH).
There is a lack of substantial evidence that androgens are effective in fractures, surgery, convalescence, and functional uterine bleeding.

Testosterone esters are less polar than free testosterone. Testosterone esters in oil Injected intramuscularly are absorbed slowly from the lipid phase; thus, testosterone cypionate can be given at intervals of two to four weeks.

Testosterone in plasma is 98 percent bound to a specific testosterone-estradiol binding globulin, and about 2 percent is free. Generally, the amount of this sex-hormone binding globulin in the plasma will determine the distribution of testosterone between free and bound forms, and the free testosterone concentration will determine its half-life.

About 90 of a dose of testosterone is excreted in the urine as glucuronic and sulfuric acid conjugates of tesrtosterone and its metabolites; about 6 percent of a dose is excreted in the feces, mostly in the unconjugated form. Inactivation of testosterone occurs primarily in the liver. Testosterone is metabolized to various 17-keto steroids through two different pathways.

The half-life of testosterone cypionate when Injected intramuscularly is approximately eight days.

In many tissues the activity of testosterone appears to depend on reduction to dihydrotestost-
erone, which binds to cytosol receptor proteins. The steroid-receptor complex is transported to the nucleus where it initiates transcription events and cellular changes related to androgen action.

Cypionate® is indicated for replacement therapy in the male in conditions associated with symptoms of deficiency or absence of endogenous testosterone.
1. Primary hypogonadism (congenital or acquired)-testicular failure due to cryptorchidism, bilateral torsion, orchitis, vanishing testis syndrome; or orchidectomy.
2. Hypogonadotropic hypogonadism (congenital or acquired)-idiopathic gonadotropin or LHRH deficiency, or pituitary-hypothalamic Injury from tumors, trauma, or radiation.

1. Know hypersensitivity to the drug
2. Males with carcinoma of the breast
3. Males with known or suspected carcinoma of the prostate gland
4. Women who are or who may become pregnant
5. Patients with serious cardiac, hepatic or renal disease

Hypercalcemia may occur in immobilized patients. If this occurs, the drug should be discontinued.Prolonged use of high doses of androgens (principally the 17-delta alkyl-androgens) has been associated with development of hepatic adenomas, hepatocellular carcinoma, and peliosis hepatis- all potentially life-threatening complications.

Geriatric patients treated with androgens may be at an increased risk of developing prostatic hypertrophy and prostatic carcinoma although conclusive evidence to support this concept is lacking.

Edema, with or without congestive heart failure, may be a serious complication in patients with pre-existing cardiac, renal or hepatic disease.

Gynecomastia may develop and occasionally persists in patients being treated for hypogonadism.

This product contains benzyl alcohol. Benzyl alcohol has been reported to be associated with a fatal “Gasping Syndrome” in premature infants.

Androgen therapy should be used cautiously in healthy males with delayed puberty. The effect on bone maturation should be monitored by assessing bone age of the wrist and hand every 6 months. In children, androgen treatment may accelerate bone maturation without producing compensatory gain in linear growth. This adverse effect may result in compromised adult stature. The younger the child the greater the risk of compromising final mature height.

This drug has not been shown to be safe and effective for the enhancement of athletic performance. Because of the potential risk of serious adverse health effects, this drug should not be used for such purpose.

General: Patients with benign prostatic hypertrophy may develop acute urethral obstruction.
Priapism or excessive sexual stimulation may develop. Oligospermia may occur after prolo-
nged administration or excessive dosage. If any of these effects appear, the androgen should be stopped and if restarted, a lower dosage should be utilized.

Cypionate® should not be used interchangeably with testosterone propionate because of differences in duration of action.

Cypionate® is not for intravenous use.

Information for patients: Patients should be instructed to report any of the following nausea, vomiting, changes in skin color, ankle swelling, too frequent or persistent erections of the penis

Laboratory tests: Hemoglobin and hematocrit levels (to detect polycythemia) should be checked periodically in patients receiving long-term androgen administration.
Serum cholesterol may increase during androgen therapy.

Drug/Laboratory test Interferences: Androgens may decrease levels of thyroxine-binding globulin, resulting in decreased total T4 serum levels and increased resin uptake of T3 and T4 Free thyroid hormone levels remain unchanged, however, and there is no clinical evidence of thyroid dysfunction.

Carcinogenesis: Animal data. Testosterone has been tested by subcutaneous Injection and implantation in mice and rats. The implant induced cervical-uterine tumors in mice, which metastasized in some cases. There is suggestive evidence that Injection of testosterone into some strains of female mice increases their susceptibility to hepatoma. Testosterone is also known to increase the number of tumors and decrease the degree of differentiation of chemically-induced carcinomas of the liver in rats.

Human data. There are rare reports of hepatocellular carcinoma in patients receiving long term therapy with androgens in high doses. Withdrawal of the drugs did not lead to regression of the tumors in all cases.
Geriatric patients treated with androgens may be at an increased risk of developing prostatic hypertrophy and prostatic carcinoma although conclusive evidence to support this concept is lacking.

Pregnancy: Teratogenic Effects. Pregnancy Category X. (See CONTRAINDICATIONS).
Nursing mother: Cypionate® is not recommended for use in nursing mothers.
Pediatric use: Cypionate® is not recommended for use in children.

Androgens may increase sensitivity to oral anticoagulants. Dosage of the anticoagulant may require reduction in order to maintain satisfactory therapeutic hypoprothrombinemia.
Concurrent administration of oxyphenbutazone and androgens may result in elevated serum levels of oxyphenbutazone.

In diabetic patients, the metabolic effects of androgens may decrease blood glucose and therefore, insulin requirements.

The following adverse reactions in the male have occurred with some androgens:
Endocrine and urogenital: Gynecomastia and excessive frequency and duration of penile
erections. Oligospermia may occur at high dosages.

Skin and appendages: Hirsutism, male pattern of baldness, seborrhea, and acne.
Fluid and electrolyte disturbances: Retention of sodium, chloride, water, potassium, calcium, and inorganic phosphates.

Gastrointestinal: Nausea, cholestatic jaundice, alterations in liver function tests, rarely
hepatocellular neoplasms and peliosis hepatic (see WARNINGS).

Hematologic: Suppression of clotting factors II, V, VII, and X, bleeding in patients on concomitant anticoagulant therapy, and polycythemia.Nervous system: Increased or decreased libido, headache, anxiety, depression, and generalized paresthesia.

Allergic: Hypersensitivity, including skin manifestations and anaphylactoid reactions.
Miscellaneous: Inflammation and pain at the site of intramuscular Injection.

Controlled Substance Class: Testosterone is a controlled substance under the Anabolic Steroids Control Act, and Cypionate® has been assigned to Schedule III,

There have been no reports of acute overdosage with the androgens.

Cypionate® is for intramuscular use only. It should not be given Intravenously. Intramuscular Injections should be given deep in the gluteal muscle.

The suggested dosage for Cypionate® varies depending on the age, sex, and diagnosis of the individual patient. Dosage is adjusted according to the patient’s response and the appearance of adverse reactions.

Various dosage regimens have been used to induce pubertal changes in hypogonadal males; some experts have advocated lower dosages initially, gradually increasing the dose as puberty progresses, with or without a decrease to maintenance levels. Other experts emphasize that higher dosages are needed to induce pubertal changes and lower dosages can be used for maintenance after puberty. The chronological and skeletal ages must be taken into consideration, both in determining the initial dose and in adjusting the dose.
For replacement in the hypogonadal male, 50-400 mg should be administered every two to four weeks.

Parenteral drug products should be inspected visually for particulate matter and discoloration
prior to administration, whenever solution and container permit. Warming and shaking the vial should redissolve any crystals that may have formed during storage at temperatures lower than recommended.

Vials should be stored at controlled room temperatures 15-30°C (59-86° F) and protected from light.

HOW SUPPLIED- Cypionate® Injection, Solution-Intramuscular-200 mg/ml is supplied in 1 ml vial.

Sidney Gordon


Cell. 561-213-7772

Ph. 866-641-CORE (2673)

Fax. 866-686-5280

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Progesterone for women

Progesterone for women

December 8, 2010

Until recently, the only commercial testosterone products available contained methyltestosterone, a synthetic form of testosterone, in dosages only appropriate for men. Current studies, however, clearly show that testosterone is also an important hormone for women. Now, because of its increased popularity, there has been a rush by both pharmaceutical companies and compounding pharmacies to meet the demand. The estradiol-testosterone combination patch Estratest® provides dosages appropriate for women, but does not contain natural, bio-identical testosterone. Presently, bio-identical testosterone can only be purchased from compounding pharmacies, formulated as tablets, capsules, creams, gels, or sublinguals.

What is Anti Aging? What can you do?

What is Anti Aging? What can you do?

December 7, 2010

Anti Aging, refers to attempts to slow down or reverse the processes of aging to extend both the maximum and average lifespan. Researchers believe that future breakthroughs in tissue rejuvenation with stem cells, molecular repair, andorgan replacement will enable humans to have indefinite lifespans through complete rejuvenation to a youthful condition.

During the process of aging, an organism accumulates damage to macromolecules, cells, tissues and organs. The maximum life span for humans is in excess of 120 years, whereas the maximum lifespan of a mouse, commonly used as a model in research on aging, is about four years. Genetic differences between humans and mice that may account for these different aging rates include efficiency of DNA repair, types and quantities of antioxidant enzymes, and different rates of free radical production.

Average lifespan in a population is lowered by infant and child mortality, which are frequently linked to infectious diseases or nutrition problems. Later in life, vulnerability to accidents and age-related chronic disease such as cancer or cardiovascular disease play an increasing role in mortality. Extension of expected lifespan can often be achieved by good diet, exercise and avoidance of hazards such as smoking.

Maximum lifespan is determined by the rate of aging for a species inherent in its genes and by environmental factors. One widely recognized method of extending maximum lifespan in organisms such as nematodes is calorie restriction. Another technique used evolutionary pressure such as breeding from only older members.

Theoretically, extension of maximum lifespan could be achieved by reducing the rate of aging damage, by periodic replacement of damaged tissues, or bymolecular repair or rejuvenation of deteriorated cells and tissues.

Our anti-aging programs and therapies are a result of years of medical research coupled with advances in biotechnology. We can now offer patients the medical treatments which combine both mainstream medicine and alternative medicine in order to provide the benefits of a vibrant lifestyle and extended lifespan. CLICK HERE TO GET STARTED!

Florida Health & Rejuvenation Center

Florida Health & Rejuvenation Center

November 29, 2010

HRT Clinic Core Institutes offers bioidentical hormone therapy, and complete hormone replacement therapy for women and men. This preventive medical approach is designed to help end the symptoms associated with menopause, andropause and hormonal imbalance.

Hormone Replacement Therapy, or HRT, is beneficial to both men and women. It is the process of replacing the hormones that your body needs to function optimally and which decline in all of us as we age. Natural Hormones, such as our human growth hormone and our testosterone are molecule by molecule exactly the same hormones present in the human body which can be replaced as your own hormones begin to decline. At our Florida HRT Clinic, your physician will assess your individual needs and work to restore these hormones during times when it is needed.

Get Started With Your Therapy Instantly!

CLICK HERE to fill out our form and begin your therapy process. You can also contact us for any required information. We want you to be fully satisfied before you begin. Your information will remain safe and confidential with us.

HCG Diet

HCG Diet

November 24, 2010

What Is It?

HCG (Human Chorionic Gonadotropin) is a hormone produced during pregnancy. One of its functions is to ensure the growing fetus receives enough nutrients to grow and develop normally. It does this by making the excess fat stored in the mother’s body available for use. The mother’s body is then able to use this fat for nutrients and energy even with limited caloric consumption. This is why a women can suffer from “morning sickness” with symptoms such as daily vomitting for months at a time and the baby does not suffer. Pregnancy is the only time HCG is found in the body.


PRO’s & Additional benefits you can get from HCG:

  1. No loss of muscle mass upon reaching your ideal weight
  2. Rebuilds the Adrenal  glands
  3. Balance your hormones
  4. Replaces good structural body fat while getting rid of the bad fat
  5. Replaces the good cushioning “fat pad” throughout the body
  6. Replaces bottom of the foot heel pad in the course of the treatment and permanently eliminates pain in that area
  7. Can help bring cholesterol and blood pressure levels down to a normal range
  8. Normalizes out of control appetite demands
  9. Gets rid of large pot belly on both male and females
  10. Eliminates double chin fat deposits
  11. Improve one’s libido
  12. Improves your “singing voice”
  13. Flushes out excess dead fat cells from the body
  14. Improvement in sleep
  15. Greatly improves energy levels
  16. Raises and resets your metabolism
  17. No exercise needed, although brisk walking daily is recommended for a healthy heart

DHEA – Dehydroepiandrosterone What you need to know.

DHEA – Dehydroepiandrosterone What you need to know.

November 23, 2010

5-Dehydroepiandrosterone (5-DHEA) is a 19-carbon endogenous natural steroid hormone It is the major secretory steroidal product of the adrenal glands and is also produced by the gonads and the brain. DHEA is the most abundant circulating steroid in humans.


DHEA Restoration Therapy

DHEA serves as precursor to male and female sex hormones (androgens and estrogens). DHEA levels in the body begin to decrease after age 30, and are reported to be low in some people with anorexia, end-stage kidney disease, type 2 diabetes (non-insulin dependent diabetes), AIDS, adrenal insufficiency, and in the critically ill. DHEA levels may also be depleted by a number of drugs, including insulin, corticosteroids, opiates, and danazol.